High C Reactive Protein Levels Are Markers For Heart Disease, Stroke and Death

If you've been following health news since the mid '90s, or watched a Bayer aspirin commercial in that span, you've probably heard about the connection between taking aspirin and reducing the risks of second or additional heart attacks or reducing strokes. While the statistical corroboration had been there (and is, indeed, enough of a reason to prescribe taking an aspirin to ward off heart conditions), it didn't actually get to the cause of the problem.

Since 2002, a lot more research has been put into tracking low-level systemic inflammation in the body. It's not always easy to detect from its physical manifestations, simply because they can be subtle and hard to spot in general, but also because most doctors don't have a good "non-inflammatory" baseline to compare a patient's condition to.

Since 2003, a broad category of proteins, called C-reactive Proteins, or hs-CRPs, have been identified as the body's internal chemical cue for the cause of low-level inflammation, which also triggers prostaglandin production through the COX-1 and COX-2 receptors. These receptors can be treated with NSAIDs, which is one of the two ways that aspirin helps reduce the risk of heart attacks. (Aspirin also helps prevent blood clotting for 4-7 days after the dose is taken, which is also a handy thing to have in preventing heart attacks.). While the triggering protein for low level inflammation may seem like nothing more than a scientific curiosity, it's actually much more useful. Now that it's been isolated, it can be screened for with a blood text. (see studies - The End of Heart Disease and Arthritis)

C Reactive Protein tests are part of the standard blood screen; to get one, the doctor will draw a few milliliters of blood from a vein, and test them with serums for protein reactions; there are standard ranges of CRP in the human body, with average levels ranging from 1 to 3 milligrams per liter of blood, elevated levels at 3.1 to 10 milligrams per liter, and levels in excess of 10 milligrams per liter a strong indicator of persistent inflammation throughout the system. High levels of CRP are strong indicators of heart disease, and increased risks of strokes and premature death. They're also considered strong indicators for both osteoarthritis and rheumatoid arthritis, as well as some liver and kidney conditions.

Low level inflammatory effects put additional stress on the lymphatic system, and make it much likelier that your immune system will turn a specific reaction against an invading microorganism or protein into a generalized system attack against soft tissues in the body; this is presumed to be the triggering cause of rheumatoid arthritis, as the body's leukeocytes start attacking the sheathes of the joins. The body can also start attacking tissues in other organs, such as the Cowpens's islets that produce insulin in the pancreas, and adrenal glands over the kidneys; early detection of elevated CRP levels make it possible to try a broader range of treatments, like enzymatic balancing or pH regulation, before falling into a harmful cycle of constant doses of NSAID-class drugs to keep inflammation down on a constant basis.

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